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SURGICAL CURES FOR FAT LOSS AND TYPE-2 DIABETES

Hilights


Book-2: Guide to Total Wellness, Vr-1.0

Fat cells are almost all created in the body by the age of two years. These cells have the ability to expand like balloons to hold more fat. Evidently the obese have not only more of such cells but also more of them storing more fat. Hence the logic for ultra sonic or cryogenic lipolysis which reduces the number of fat cells and thus the fat stored. However if the diet is not properly controlled thereafter, the body fat can easily return to previous levels by each remaining fat cell storing more. These procedures are new and the process of elimination of the fat after bursting the fat cell is to be better understood. You may resort to such treatment for stubborn fat deposits, but do so in small stages (Also see – ‘Non Conventional Treatments & Folk Remedies’). Surgical liposuction should these days be a last resort option and only when so advised by your Physician.

Researchers using data from the National Health and Nutrition Examination Survey published in ‘The Lancet – Diabetes & Endocrinology, have noted that it is the fat cells pigment epitheliums derived factor that makes muscle and liver ignore insulin, triggering a chain of events and interactions that lead to the development of Type-2 diabetes. Hence the more fat cells a person has, the less sensitive they become to insulin, which then calls for a greater amount for insulin to maintain the body’s regulation of blood-glucose, thus making it difficult for the obese to control diabetes. Of course other studies have questioned BMI being used as a definer. Hence the question remains how obese is obese?

Type-2 Diabetes was, till recently, thought to be chronic, progressive metabolic disease caused by high prevalence of obesity. However, it is today recognized as a reversible dietary disease caused by insulin resistance which is itself linked to greater susceptibility due to the ever increasing consumption of sugar and refined carbohydrates in our diet.

Also the excess consumption of fructose, the main sugar in fruits, is chemically similar to glucose and carries similar number of calories, but the two are processed differently by the body. Fructose also tastes sweeter and seems to make people hungrier. Cells in the body regulate the amount of glucose they use to make energy, while fructose is generally metabolized in the liver. Fructose does not drive the body to produce insulin, a hormone that plays an important role in curbing appetite and making food less appealing. Increased consumption of Fructose is considered to be a major contributor to the prevalent obesity and diabetic epidemic.

Type-2 Diabetes is a worldwide epidemic with many contradictory findings about its real causes and effects and attempting to achieve smooth normal blood sugar level readings with just conventional treatments, comprising of eating less, exercising more and taking oral medication and insulin does not work. As these do not effect what research is increasingly showing to be the main cause, the excess sugar and carbohydrate consumption in our diet. The intestinal hormones released by our Gut micro-flora have a striking effect on the pancreas and on insulin secretion.

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